| Papers [1-14] of 100 :: [Page 1 of 8] | | Go to page : 1 2 3 4 5 6 7 8 —> | Search results on "MAD COW DISEASE": |
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Mad Cow Disease, 2009.
An analysis of the reality vs. perception of mad cow disease in the U.S., Germany and the Netherlands.
1,517 words (approx. 6.1 pages), 8 sources, APA, £ 31.95
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Abstract
The paper considers the official, public and economic responses of the mad cow scare on three nations; the U.S., Germany and the Netherlands. The paper reveals that in spite of the dramatic response taken by the governments, the research shows how America and Europe are moved more by panic than by fact. The paper asserts that the dangers of mad cow disease to human beings have been grossly exaggerated, creating an unrealistic public fear and the destruction of an industry. The paper maintains that the greatest preventative measure for mad cow disease is the proliferation of public and official understanding of what it is and how it is caused.
From the Paper
"Recent years have witnessed an array of global food-borne health scares which are related to practices of careless governmental oversight, haphazard agricultural standards and increased free trade activities between industrialized nations and those developing nations which are either unable or unwilling to devote the necessary resources to administrative regulation. The outcome of these conditions has been an apparent invasion of certain advanced economies and societies of terrifying disease strains and the public panics there associated. Among those scares most publicly aired by the news would be those relating to Bovine Spongiform Encephalopathy, which is more commonly known as 'Mad Cow Disease' or 'Mad Cow's Disease.' Between the years of 2001 and 2004, an outbreak of cases in a host of industrialized nations would spark the biggest public panic to date, prompting publics in said nations to abstain from beef, prompting such industries to experience devastating economic decline, inciting public airwaves to dedicate considerable focus to the subject and inspiring governmental leaders to draw up and pass legislation concerning the prevention of the disease spread and the prohibition of practices believed to cause it."
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Mad Cow Disease, 2004.
An analysis of anticipated increases in business stock prices due to impact of mad cow disease on U.S. cattle.
1,816 words (approx. 7.3 pages), 5 sources, MLA, £ 36.95
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Abstract
The paper intrinsically studies the consequential affect and aftermath of ?mad cow disease?, which has led to the introduction of substitutes like pork and other meats in the market. It further studies the changing trends that many of these companies have incorporated in attempt to sustain and raise their profits from these substitutes. It looks at how studies show that integrating certain changes to the companies marketing mechanism and strategies on the eve of ?mad cow disease? have been successful, not only to replenish the sales figures, but also in incurring profits.
From the Paper
"The Other areas where consolidation occurred were the packing and retail industries. In an attempt to regain their market hold various leading food giants are vying to increase their sales and are introducing new trends. The four major companies, which followed the same suit, were Smithfield Foods, IBP, Swift (Conagra) and Excel (Cargill). In 1998, the four-firm concentration level was 56.3% of the packing sector. They contributed to the slaughter of more than half of the nation's hogs. Now Smithfield is the world's largest pork producer and kills an estimated 20% of the United States' hog supply. It considered itself as an integrated food company. Eventually owned the third largest hog producing firm in the US and also had been tightly coordinating with other large producers."
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Mad Cow Disease and McDonald's, 2002.
A look at the efforts made by McDonald's to educate the public on the dangers of Mad Cow Disease .
1,150 words (approx. 4.6 pages), 5 sources, £ 27.95
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Abstract
This paper assesses the issue of advertising safety precautions for Mad Cow Disease from the position of a campaign manager for McDonald's customer service division. This paper will take the position that the public would remain loyal to McDonald's after efforts are made to educate them concerning mad cow disease.
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Mad Cow Disease: Governmental and Media Culpability, 2002.
A discussion of the discovery of mad cow disease, to the present, and the role the media/government has played in control, prevention, and the dissemination of information.
2,000 words (approx. 8.0 pages), 8 sources, MLA, £ 40.95
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Abstract
Examines the treatment of Mad-cow disease in the media, and by the government.
Discusses the following topics
Bovine Spongiform Encephalopathy and Scrapie
What Transmissible Spongiform Encephalopathies Do
Two Theories on the Action of Transmissible Spongiform Encephalopathies
British Government Admits Mad Cow Disease Exists (1987)
Testing Results by the British government MAFF
Culpability of the British Medical Society
The Spread of CJD
Steps Taken by the British Government
From the Paper
"Where the industry has gone wrong is in listening to the PR people. The mad-cow epidemic is not an information management issue?it is a disease that will not go away.? These words are from an interview with Dr. Pringle, a member of the Sperling Biomedical Foundation, and the founder of www.mad-cow.org. I believe that they highlight what has been fundamentally wrong about the treatment of mad-cow disease by the British government and the British media. Several grave errors have been made about the treatment of this issue, and they continue to have repercussions today, as we explore America?s mad-cow incidents, and rising problems in Europe, Asia, and South America. At the time this paper was written, mad cow disease had been confirmed in domestic cattle in Belgium, France, Ireland, Liechtenstein, Luxembourg, the Netherlands, Portugal, Switzerland, Spain and Germany."
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Mad Cow Disease, 2009.
An analysis of Germany's response to an outbreak of mad cow disease.
1,199 words (approx. 4.8 pages), 15 sources, APA, £ 26.95
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Abstract
The paper discusses the efficiency of the German governmental agencies and beef industry in estimating the risk of mad cow disease negatively affecting German consumers of beef during the years 2001 to 2004. The paper further discusses how the German governmental agencies and industry managers performed risk management strategies and policies through the selection and implementation of appropriate measures after the outbreak of mad cow disease in Germany in 2000 and until 2004. In addition, the paper looks at how the risk communication was conducted in relation to the true possibility that humans can contract Creutzfeldt-Jacob Disease (CJD), a variant of BSE.
Outline:
Objective
Introduction
Perceived Risk Analysis
Rational Problem-Solving
Food Safety Regulation Reform
Research Risk and Assessment
Food Industry Supports Government
Summary and Conclusion
From the Paper
"The work of Grannis, Green and Bruch (2004) entitled: "Animal Health: The Potential Role for Livestock Disease Insurance" state that "animal disease can cause significant production losses and a reduction in livestock receipts." Grannis, Green and Bruch state that an opportunity development exists in assisting the management of livestock disease risks and one example is Germany. "Evidence from Germany demonstrates that both alternative value and consequential loss policies for livestock producers can be developed and can work in parallel with government indemnity programs. In Germany, indemnity payments are made based on predetermined and published payment schedules. These schedules are capped by maximum values that do not represent the value of superior commercial animals. Policies are available to insure the difference between the value indemnified according to the published government schedule and the value of the animal under regular, nondisease market conditions".
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Mad Cow Disease, 2004.
This paper discusses mad cow disease, a virulent cattle disease, which led to the destruction of 180,000 livestock in the United Kingdom and other European countries and plunged other major cattle-producing nations into a global panic.
1,500 words (approx. 6.0 pages), 8 sources, MLA, £ 31.95
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Abstract
This paper explains that mad cow disease is bovine spongiform encephalopathy, or BSE, a fatal brain disorder in cattle caused by a still-unknown agent. The author points out that U.K. reports also listed other animals as having been infected by TSEs, including domestic cats, mice, hamsters, goats, mink, monkeys, pigs, and some exotic species of the cat family. The paper reports that there is also suspicion that mad cow can be spread through human blood; therefore, the American Red Cross bans blood donations from people who have lived abroad, especially from those who spent as few as three months in the UK between 1980 and 1996.
From the Paper
"The disease spread to cattle in other countries through infected animal feed UK exported to them within that decade. These countries in Europe alone included Belgium, Canada, the Czech Republic, Denmark, France, Germany, Greece, Ireland, Italy, Luxembourg, Liechtenstein, the Netherlands, Portugal, Spain and Switzerland (Lohn), which were reported to have at least one infected cow each. The UK also supplied animal feed to South Africa and non-European countries at that time, extending the reach Mad Cow beyond Europe and making it a global health scare."
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U.S. Fast Food and Mad Cow Disease, 2004.
Examines mad cow disease and how fast food companies are responding.
690 words (approx. 2.8 pages), 2 sources, APA, £ 14.95
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Abstract
This paper examines a 2004 Wall Street Journal article on mad cow disease, and how companies, especially fast food companies, are responding to the possibility of tainted meat. It uses the example of McDonald's.
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Mad Cow Disease, 2007.
An analysis of the epidemiology, pathophysiology and prevention for Mad Cow Disease (known as Creutzfeldt-Jakob disease in humans).
1,445 words (approx. 5.8 pages), 10 sources, MLA, £ 29.95
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Abstract
This paper discusses Mad Cow Disease, otherwise known as Bovine Spongiform Encephalopathy or as Creutzfeldt-Jakob disease in humans. It discusses the history, background and pathophysiology of the disease. It then goes on to describe prevention and treatment options for the disease, concluding that there are no effective treatments available. The paper concludes by discussing nursing and collaborative care for patients with the disease.
Table of Contents:
Epidemiology
Pathophysiology
Prevention and Treatment
Differences in Classic CJD and Variant CJD
Nursing and Collaborative Care
From the Paper
"Variant CJD has a median age at death of 28 years. The duration of the illness is approximately 13-14 months. Some of the clinical signs and symptoms include: prominent psychiatric/behavioral symptoms, painful dyesthesiasis; and delayed neurologic signs. The "Pulvinar sign" on an MRI is present in greater than 75% of most of the Variant CJD. The presence of "florid plaques" on neuropathology is usually in large numbers and easily detected. The agent is readily detected in the lymphoid tissue. There is a marked accumulation of protease-resistance prion protein noted in the brain. This is why the Variant CJD is much more easily detected because it shows up on more tests."
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Mad Cow Disease in the Netherlands, 2009.
An analysis of the Dutch government's response to the outbreak of mad cow disease.
1,625 words (approx. 6.5 pages), 11 sources, APA, £ 33.95
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Abstract
The paper discusses how, although the media in the Netherlands inflated the real threat of bovine spongiform encephalopathy (BSE) to human beings, the Netherlands still failed to respond appropriately to the sense within the public that its government had not protected it properly from dangerous foods. The paper thus highlights the need for the government to take the threat of BSE seriously, not as an immediate danger to the European public, but as a way of sustaining the public's confidence in the regulation of food safety.
From the Paper
"The risk of human contraction mad cow disease is perceived to be high in places where its early form can be detected in livestock. Known in this context as Bovine Spongiform Encephalopathy (BSE), consumption of meat thereby contaminated is believed to be a way of contract the fatal neurologically degenerative human disease called Creutzfeldt-Jakob (vCJD). Thus, when reports surfaced of contamination found in meats allegedly originating on Dutch farms, the outcome was a dramatic response from various governments within Europe, amongst EU agencies and sectors of various national publics, all of which responded to the threat with an immediate forbearance from meat. The question must be posited, however, with regard to its dramatic spread in the public discussion forum, as to whether the response has thus far been appropriate or properly measured from within the Dutch government."
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Mad Cow Disease, 2006.
An overview of this brain disease which affects cattle and humans.
857 words (approx. 3.4 pages), 4 sources, MLA, £ 19.95
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Abstract
Mad Cow Disease is a brain disease of cattle, which was first identified in the United Kingdom in the mid-1980s. Although not as widespread as some other livestock diseases, such as hoof-and mouth-disease, the Mad Cow Disease has attracted a lot of publicity because of its apparent apparent ability to transmit to humans, the fact that there is no known cure for the disease and the horrifying nature of the brain decay it causes. This paper explains the cause of the disease, how it affects the organisms, how it is transmitted from one organism to another, and the ways in which it could be transmitted to humans. The paper also discusses the human equivalents of the disease.
Paper Outline:
Cause of the Disease
How the Disease Affects the Organism
How is the Disease Transmitted from one Organism to Another
The Ways in Which it Could be Transmitted from Cows to Humans
Human Equivalents of the Disease
Works Cited
From the Paper
"This theory about the spread of disease is based on the observation that incidences of the disease have mostly been found in the UK where feeding of meat and bone meal to cattle was most common. Although other European countries also fed meat and bone meals to cattle as a protein supplement, the British laws about high temperature sterilization of the protein meal were relatively lax in order to keep meat prices competitive."
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Mad Cow Disease, 2005.
An overview of the causes, treatment and prevention of Bovine Spongiform Encephalopathy (BSE).
1,438 words (approx. 5.8 pages), 7 sources, MLA, £ 29.95
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Abstract
This paper examines how Bovine Spongiform Encephalopathy (BSE) is a neuro degenerative disease in a class of diseases known as Transmissible Spongiform Encephalopathies (TSE's). It looks at how BSE is caused by prions, which are related to several other TSE's and how it has been a major concern with the economy and human and animal health worldwide together with political policies. It also explores how testing, diagnosis and prevention of BSE and other TSE's are still under investigation.
From the Paper
"vCJD also affects humans, but differs from CJD. Symptoms include depression, mood swings, anxiety, withdrawal, hallucinations, delusions, parasthesia ("pins and needles"), headaches, cold extremities, lack of coordination, loss of memory, loss of concentration, inability to talk or move, and results in death after 7-24 months, averaging 14 months.(7) It affects people ages 8-14.(7) 156 confirmed or probable cases have been reported worldwide as of April 2004. 146 of these cases were reported in Great Britain, 6 in France, and one in each Italy, Ireland, United States, and Canada. (7)Treatment for this disease includes chlorpromazine and quainacrine, but only experimentally."
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Mad Cow Disease, 1996.
Bovine spongiform encephalopathy. Causes, symptoms, effects on humans, British govt. & public reaction to outbreak, economics.
1,350 words (approx. 5.4 pages), 7 sources, £ 29.95
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From the Paper
"Many people claim that they want less government. In the case of Great Britain's problems with "mad cow disease" this is exactly what they got. Yet few people can be pleased. On March 20, 1996, the United Kingdom Health Minister announced that ten relatively young people had died of a very rare brain disease, Creutzfeldt-Jakob disease (CJD), that generally attacks only a very few elderly people. The minister reported that "the most likely explanation at present is that these cases are linked to exposure to bovine spongiform encephalopathy (BSE) or mad cow disease (Bonte-Friedheim A10). This is tragic and alarming but it is also outrageous. BSE has been a serious problem for Britain for a long time yet "for a decade [the Conservative government] has dismissed concerns raised by some scientists that humans could contract the disease by eating the meat of diseased..."
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Prion Disease in Humans, 2005.
An overview of Mad Cow Disease and how it has benefited the medical world.
1,636 words (approx. 6.5 pages), 5 sources, APA, £ 33.95
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Abstract
Ever since Mad Cow Disease sparked terror in international headlines, the public has been concerned about eating beef. Mad Cow Disease is not, however, the first or only prion-caused disease. Several others, including Creutzfeldt-Jakob Disease (CJD) and kuru in humans and a host of agricultural animal disorders join Mad Cow in both cause and effects, the major effect being horror. This paper argues that, while the presence on the planet of a disease that literally lays waste to human brain tissue is frightening, there are two probabilities that can be seen as reasons not to be terrified. Instead, people could be hopeful that research sparked by the publicity surrounding Mad Cow Disease will eventually bring answers to such sociologically and medically devastating diseases as Alzheimer's Disease, and other lesser known but just as deadly diseases such as Lou Gehrig's Disease. The paper shows that these two probabilities are first, that prion-caused diseases are much less prevalent than media hype would indicate and, second, that a cure or vaccination is relatively certain to be discovered in the near to medium term.
Paper Outline:
Introduction
Methods
Discussion
Conclusion
References
From the Paper
"Questions have arisen regarding why these particles are not attacked by the body's defense mechanisms, and the answer, Mahy noted, is that they are so odd, they are simply not recognized and therefore cannot be neutralized. Prions contain no nucleic acid and are therefore extremely " extremely resistant to inactivation by chemical or physical interventions that would inactivate viruses" (Mahy 1998) because there is no vector through which to deliver the 'poison'. Even more so than viruses, then, it is reasonable to wonder whether prions are, in fact, alive. And if they are not alive, then how can they infect an organism? Do they replicate like bacteria and viruses, or what?"
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Prions and Prion Diseases, 2001.
A discussion of the various aspects of prions and prion diseases.
1,307 words (approx. 5.2 pages), 4 sources, £ 27.95
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Abstract
This paper examines the novel mechanism of disease transmittal in prions as well as the diseases themselves (mad cow disease and Creutzfeld-Jacobs disease). Functions of normal prions are also discussed. The paper also examines how prions are devoid of nucleic acids, and thus carry no information other than their tertiary structure.
From the Paper
"Knockout mice survived up to their 70th week, and then they began to display signs common with brain disease, which included a loss of motor coordination. Curiously, the symptoms shown by these knockout mice were similar to those found in many cases of prion diseases."
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